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08 Jun 2013

The Secret to Maintaining Cardiovascular Health

Three 1998 Nobel Laureates, building on decades of scientific research, have identified the endothelium factor and linked it to the health and vascular age of your arteries, veins and capillaries.
What is the Endothelium?
It is a single layer of selectively permeable cells that line the entire cardiovascular system and exerts tremendous control over the flow of blood. Lumped together, the endothelium is the largest organ of the body, weighing as much as the liver. Extraordinary new studies clearly indicate that damage to the endothelial lining is the trigger of current markers of vascular dysfunction, including plaque formation and inflammation.
The endothelium protects the integrity of the vascular system, providing a teflon lining that prevents cells from sticking to our vascular walls. It accomplishes this through the production of Nitric Oxide (NO).

The Role of Nitric Oxide (NO)
As nitric oxide levels declines, so does endothelial function. As endothelial function declines, so does the elasticity and integrity of our vascular system. This continuous breakdown of the vascular system has tremendous ramifications on your health.
Just think about the capabilities of large arteries, medium sized arteries and our peripheral veins, the capillaries that carry nutrients to the furthest extremities of the body:
 Every 60 seconds, our vascular system distributes no less than 5 quarts of life sustaining blood, an extraordinary 1800 gallons per day.
 The vascular system also distributes oxygen from the lungs, nutrients from the intestines, hormones from the brain and glands and white blood cells, the foundation of the body’s ever-vigilant immune system.
 All waste is removed from the body and delivered to the liver and kidneys for breakdown and excretion.
 The vascular system can signal the blood to clot when there is a cut. It can control the diameter of the smallest capillary, increasing flow to muscles during exercise and decreasing flow to the skin when the body reacts to fear.
 The small vessels dilate and constrict to direct the flow. The large arteries respond to the hearts rhythm, expanding with each beat and relaxing between beats.

Arteriosclerosis
Under microscopic observation, our vascular system reveals the effect of plaque buildup in the arteries – quite literally blemishes that have earned the name arteriolosclerosis.
 A rupture or damage to the endothelium attracts the “bad cholesterol” (LDL-C) which sticks and oxidizes. . .and attracts white blood cells. This powerful army generated by the immune system engulfs oxidized cholesterol until, engorged, they swell into foam cells.
 If the endothelium does not recover, LDL-C and Foam Cells form a pustule, much like a pimple, which signals for more white blood cells and a powerful antioxidant, super oxide ion.
 When the abscess pops, it releases puss and causes a blood clot that can lead to heart attack or stroke.
 An aging vessel wall looks like skin scarred with acne!

Nobel Prize winning research shows us how we can restore endothelial function by boosting NO production.
Nitric oxide slows plaque growth and suppresses arteriosclerosis, keeping vessels pliable and elastic.
Nitric oxide reduces the stickiness of the endothelium and prevents white blood cells and platelets from clinging to the vessel wall.
 Nitric oxide calms the cells flowing through the vessel and, thereby protects the vessel wall. Even when blood cholesterol is high, if the vessel is producing sufficient amounts of NO, the vessel will be protected from the development of plaque.
 Nitric oxide melts away or shrinks plaque that is already there!
 Nitric oxide regulates oxidative enzymes (ADMA) in the cell, preventing oxidation and the free radical damage it generates.
 Nitric oxide has been shown to relax blood vessels and lower blood pressure.
This research notable of the coveted Nobel Prize in Medicine (1998) is changing the landscape of cardiovascular health and adding credibility to the right mix of nutritional supplements

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04 May 2013

Arginine & Hypertension

Hypertension is a major healthcare problem afflicting nearly 50 million individuals in the United States. Despite its strong causal association with cardiovascular disease complications including myocardial infarction, heart failure, and stroke, the majority of patients with hypertension do not achieve optimal blood pressure control. The prevalence of hypertension is expected to increase with the aging population, growing obesity epidemic, and rising incidence of metabolic syndrome.

Endothelial dysfunction and reduced nitric oxide (NO) bioactivity represent prominent pathophysiological abnormalities associated with hypertensive cardiovascular disease. Individuals with hypertension exhibit blunted epicardial and resistance vascular dilation to endothelium-derived nitric oxide (EDNO) agonists in the peripheral and coronary circulation that likely contributes to mechanisms of altered vascular tone in hypertension. The amino acid arginine serves as the principal substrate for vascular NO production. Numerous studies, though not uniformly, demonstrate a beneficial effect of acute and chronic arginine supplementation on EDNO production and endothelial function, and arginine has been shown to reduce systemic blood pressure in some forms of experimental hypertension.

Arginine dilates blood vessels, reduces blood pressure, mimics the activity of nitroglycerine, and produces nitric oxide (NO). Arginine contributes to normal blood vessel function. Congestive heart failure often reveals blood vessels that fail to dilate in response to certain drugs, a sign that the inner blood vessel wall, or endothelium, is compromised. Arginine produced a fourfold increase in blood vessel dilation (Hambrecht et al. 2000). Doses of 5.6-12.6 grams of arginine increased blood flow to the extremities by 29% (Rector et al. 1996). The effectiveness of arginine relates to its ability to directly create NO, a vasodilator produced in endothelial cells by the enzyme nitric oxide synthase (Brunini et al. 2002). Nitric oxide counteracts the vasoconstrictive effects of adrenaline and maintains vascular elasticity. Arginine increases nitric oxide, but hypertension, hyperhomocysteinemia, diabetes, and smoking decrease it.

Arginine is frequently used as a treatment for hypertension. A defect in nitric oxide production is a possible mechanism of hypertensive vascular disease (Campese et al. 1997). Some cardiologists recommend arginine over nitroglycerine, since the two substances appear to replicate a similar vascular function, that is, the ability to relax smooth muscles and dialate blood vessels.

The inside of blood vessels is lined with a layer of single cells called the endothelium. Among other functions, the endothelium produces nitric oxide that serves to relax (vasodilate) the blood vessels so as to facilitate the flow of blood. It is now generally accepted that many heart problems involve a dysfunction of the endothelial vasodilator mechanism. Antioxidants, estrogen, exercise, folic acid, and fish oils can in a number of cases, reverse this dysfunction. Now researchers at the Stanford University School of Medicine report that supplementation with the amino acid arginine is highly effective in reversing endothelial dysfunction. It has been established that arginine is the precursor for endothelium-derived nitric oxide (EDNO). EDNO, in turn, is a potent vasodilator and inhibits platelet aggregation and the adherence of circulating blood cells to blood vessel walls. Arginine administration, either orally or intravenously, has been found useful in preventing and reversing atherosclerosis, in increasing coronary blood flow in heart disease patients, in alleviating intermittent claudication, and in improving functional status of heart failure patients. Arginine infusions have been found to lower blood pressure and to inhibit restenosis (reclosing of arteries) after balloon angioplasty. The most common used dosage of arginine is between six and thirty grams per day.

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